TEA 67

Pack Weight: 50g .

Price : 25 $ USD

Composition :

Foeniculum vulgare / Fruits
Malva sylvestris / Flowers
Thymus serpyllum / Leaves
Salvia officinalis / Leaves
Cnicus benedictus / Leaves

TEA 67 : Herbal infusion made from natural medicinal plants with no known side effects.

Indications  :

– Gastric infections.
– Gastrointestinal inflammation.

Benefits :

– Reduces inflammation and irritation of the gastric and rectal mucosa.
– Relieves spasms, bloating, and associated belching.
– Supports the natural restoration of gut microbiota balance.

Enhanced Effect:

The effect of TEA 67 is more beneficial when taken in combination with the following, depending on the condition:
– Diarrhea: Infusion 10.
– Indigestion or digestive disorders: Infusion 4.
– Irritable bowel: Infusion 16.
– Belching, functional dyspepsia: Infusion 38.
– Constipation: Infusion 9.
– Bloating, intestinal gas: Infusion 7.

The Stomach :

The stomach serves as a reservoir and is the site of breakdown of solid food into chyme. Muscular contractions mix and churn the ingested food.
Chemical digestion involves gastric glands secreting gastric juices (2–3 liters per day), including hydrochloric acid and digestive enzymes produced by gastric cells. These are mixed with the food before the chyme is passed into the intestine. The stomach typically empties 4 to 6 hours after a meal.

Stomach Pathologies :

Aerophagia, gastroesophageal reflux, heartburn, gastroenteritis, gastric ulcer, hiatal hernia, stomach cancer, etc.

Gastric Ulcer :

A gastric ulcer is inflammation of the stomach lining, often due to Helicobacter pylori (H. pylori), a bacterium that invades and destroys the protective mucus layer, leaving the gastric wall exposed to acidity and prone to lesions.

Gastritis :

Inflammation of the stomach lining, characterized by heartburn and/or difficulty digesting. Causes include alcohol, NSAIDs, H. pylori infection, severe stress, or anxiety.

1. pylori Infection :
2. pylori is a Gram-negative pathogenic bacterium responsible for gastritis, gastroduodenal ulcers, gastric adenocarcinomas, and low-grade gastric lymphomas. It is primarily found in the antrum and promotes secretion of gastrin and somatostatin.
   Patients may be predisposed to duodenal and prepyloric ulcers. Infection of the gastric corpus may lead to atrophic gastritis and reduced acid production, predisposing to gastric ulcers and adenocarcinoma.
   H. pylori produces ammonia, allowing it to survive in acidic environments, and secretes cytotoxins and mucolytic enzymes (protease, lipase), which contribute to ulcerogenesis.
   Infected individuals are 3 to 6 times more likely to develop gastric cancer.

Symptoms :

– Severe gastric discomfort or necrotic-like sensation (dyspepsia), sometimes relieved by food.

– Nausea.

– Vomiting.

– Feeling of fullness in the upper abdomen.
Note: Gastric inflammation may be asymptomatic. Any injury or weakness of the mucosa allows gastric acid to damage the stomach lining.

Causes and Risk Factors

– Bacterial infection.

– Regular use of analgesics.

– Aging (thinning of mucosa).

– Excessive alcohol intake.

– Psychological stress.

– Severe trauma or burns.

– Surgery.

Autoimmune Gastritis :

Autoimmune atrophic metaplastic gastritis occurs when the body attacks its own gastric cells (e.g., in Hashimoto’s thyroiditis or Type 1 diabetes). Antibodies target parietal cells and their components, including intrinsic factor and H⁺/K⁺ ATPase proton pump.
Hypochlorhydria leads to G-cell hyperplasia and elevated gastrinemia, potentially resulting in carcinoid tumors.

Other Associated Conditions :

– HIV.

– Crohn’s disease.

– Parasitic infections.

Recommendations :

– Eliminate harmful factors: smoking, alcohol, NSAIDs

– Protect the gastric lining:

• Eat smaller, more frequent meals.
• Follow a diet rich in fiber and antioxidants.
• Avoid combining fats with carbohydrates.
• Reduce refined foods.
• Limit irritants to the gastric lining.
• Increase intake of anti-inflammatory foods.

– Support overall well-being.

Pharmaceutical Treatments :

– Gastric protectants.

– H2-receptor antagonists.

– Proton pump inhibitors (PPIs).

– Antibiotics for H. pylori infection.

– Absorbable and non-absorbable antacids (short-term use).

– Prostaglandins (which reduce cAMP to strengthen mucosal defenses).

– Mucus-producing cells form a protective film on inflamed areas, shielding them from acid, pepsin, and bile salts.