TEA 32

Pack Weight: 50g .

Price : 25 $ USD

Composition :

Ginkgo biloba / leaves
Rosmarinus officinalis / leaves
Salvia officinalis / leaves
Cinnamomum aromaticum / bark

TEA 32 : Herbal infusion made from natural medicinal plants, with no side effects.

Indications  :

– Memory disorders.
– Concentration difficulties.
– Memory decline.
– Cerebral circulation disorders.
– Circulatory failure.
– Tinnitus, dizziness due to inner ear inflammation.
– Prevention of neurodegenerative and age-related diseases.
– Prevention of diabetic neuropathy.

Benefits :

– Stimulates memory and prevents cognitive decline.
– Improves concentration and attention during study and work.
– Enhances cerebral blood flow.

Reinforced effect:
The effect of TEA 32 is enhanced when combined with:
– Fatigue or asthenia → TEA 8.
– Insomnia → TEA 24.
– Stress → TEA 25.

The Functioning of Memory :

Memory is a biological process that allows the storage, retention, and retrieval of information. Without memory, we would be deprived of identity, expression, knowledge, reasoning, and even the ability to project ourselves into the future. Memory is one of the essential components of intelligence, with each memory being associated with a specific and remodelable neuronal network.

To memorize information, the brain performs three successive operations:

– Encoding, the learning phase, which depends on emotional state and degree of attention.

– Storage, during which the brain organizes, labels, and classifies the information.

– Retrieval, which consists of recalling and reproducing the stored information.

A memory of an event is influenced by impressions or images that reflect our interpretation of the event and our personal history, to which sensory signals are added. Memory encompasses perceptions, reactions, goals, emotions, imagination, and thought processes.

At the brain level, memorization occurs through mechanisms of consolidation and maturation of synaptic plasticity, involving modifications of neuronal connections. Each of the approximately 100 billion neurons receives thousands of synapses from many other neurons, leading to an estimated quadrillion synaptic connections. This processing capacity enables thought, feeling, and action.

If a neuron is strongly activated, it can stimulate a target neuron via one or several synapses. Synaptic plasticity plays a crucial role in learning and memory. A memory corresponds to a variation in electrical activity within a specific circuit formed by several neurons. Its formation relies on the strengthening or creation of temporary synaptic connections, stimulated by proteins such as glutamate, NMDA receptors, or syntaxin, which modulates glutamate release.

Memory consolidation depends on the availability of cellular mediators in the involved neuronal network in the hours following encoding. Strengthening or weakening synaptic connections determines whether a memory is consolidated or forgotten. Morphologically, synaptic plasticity is associated with changes in synapse shape and size, conversion of silent synapses into active ones, and the growth of new synapses.

Long-term memory maintenance depends on modifications in the elimination or renewal kinetics of certain mediators, such as Protein Kinase M Zeta (PKMζ), which self-replicates and resists inhibition.

Types of Memory :

– Short-term memory (working memory): essential for daily activities (planning, counting, coding), lasting from 0.5 seconds to 10 minutes. It relies on neuronal activity in the prefrontal and temporal cortex.

– Plural memory: involving multiple prefrontal cortical areas, critical for reasoning, planning, and performing specific mnemonic operations.

– Long-term memory: where information is stored depending on its abstraction level, subdivided into:

• Implicit (non-declarative) memory: related to motor, verbal, and cognitive skills. Past experiences facilitate performance unconsciously, without deliberate recall. This includes automatisms and procedural skills.
• Explicit (declarative) memory: involving consciously retrievable information, subdivided into:
  • Semantic memory: storage of facts, concepts, and general knowledge, organized in semantic networks. Retrieval time depends on the distance between nodes (concepts). Accessibility may fail, but information is not necessarily lost.
  • Episodic memory: autobiographical memory of personal past events, also enabling projection into the future.
  • Sensory memory: brief retention of perceptions (visual, auditory, tactile, gustatory, olfactory).
  • Eidetic (photographic) memory: hypothesized ability to recall large amounts of sensory information for up to 30 seconds.

Memory and Aging :

With aging, synaptic plasticity decreases, and connection modifications become more transient, explaining difficulties in retaining information. Cognitive reserve corresponds to the efficiency of neuronal networks in task execution and the brain’s ability to recruit compensatory networks in case of pathology. It depends on education, intellectual stimulation, social interaction, genetics, diet, physical activity, and sleep quality.

Emotions modulate memory encoding by enhancing attention. Recall is proportional to emotional intensity, with positive emotions having a protective effect. Conversely, traumatic experiences can lead to dissociative amnesia, an unconscious adaptive defense.

Forgetting is a physiological process essential to brain function, preventing overload of neural circuits. However, it becomes pathological when it involves entire areas of semantic or episodic memory.

Post-traumatic stress disorder (PTSD) is characterized by persistent, emotionally intense memories due to glucocorticoid release in the hippocampus during trauma.

Memory Disorders

Persistent amnesias may result from:

– Traumatic brain injury.

– Stroke (ischemic or hemorrhagic).

– Brain tumors.

– Neurodegeneration (e.g., Alzheimer’s disease).

Reversible, less severe memory disorders may result from:

– Depression.

– Stress, anxiety, or fatigue.

– Traumatic events (e.g., bereavement).

– Side effects of hypnotics or anxiolytics.

– Drug use.

Biological origins of amnesia involve neurotransmitter deficits or reduced brain network connectivity.

– Semantic dementia: episodic memory remains, but patients progressively lose word and factual knowledge due to temporal lobe atrophy.

– Episodic memory impairment in Alzheimer’s disease: linked to frontotemporal atrophy.

– Transient global amnesia: sudden-onset memory loss lasting 6–8 hours, with preserved remote memories.

Future Perspectives :

Humans possess a prospective memory that enables planning of future scenarios, composed of thoughts, images, and actions. Modern life has introduced external memories, through digital storage devices, altering cognitive organization.

Emerging fields such as memory prosthetics and optogenetics (activation or inhibition of specific neuronal groups) are being explored for therapeutic purposes in psychiatric and neurological disorders.

Optimizing Memory

Improving memory requires:

– Attention and prioritization of information.

– Mental repetition (silent or aloud).

– Avoidance of routine.

– Adequate sleep.

– Brain oxygenation through physical activity.

– Healthy diet.